Translation

[Reza1969]

If you have a short text (in Persian or in English) and have a problem translating it, you can post it here and your friends can help you with it. Please do not post long texts.

[silver]

dear reza ... thanks for helping and improving our english knowledge..and congratulations for your EFl moderator post.. I have request for translating the below words ...I need them for a furniture web design

مبل استيل
2- مبل راحتي
3- بوفه ويترين
4- آينه کنسول
5- دراور
6- جلومبلي (عسلي)
7- پارتيشن
8- پيراهن مبل
9- رويه کوبي
10- ورق (mdf و فرميکا و ...)
11- ثرمه
12- معرق
13- برنز
14- منبت کاري
15- مصنوعات فلزي
16- جاکفشي

thanks a lot

[Ship Storm]

O Arab of the desert, I fear thou wilt not reach the Kaba Because the road on which thou travelest leads to Turkestan. Who is the poet of this couplet?

[ghaffar]

دقیقا این جملات رو بیان میکنه. خیلی واضحه که.
این میشه: یه دوستم. عملا یه دوست مال من.
Listen and repeat: A friend of mine
::: یک توضیح:::
یه اشتباه گرامری کوچک هم داری:
بعد از to بايد فعل ساده به كار بره يعني:
to learn

Persian post as usual

[hamed79]

بچه ها كارم گيره اگه كسي ميتونه سريع بهم كمك كنه لطفا .
من ترجمه اين متن رو لازم دارم با يه عالمه عجله.
3 قسمتش كردم اگه هر كدوم از دوستان ميتونست مثلا يك قسمتو بگه و دوستاي ديگه جاهاي ديگشو .
ببخشين تو رو خدا فقط تا فردا لازم دارم

**01*****

As indicated in the text, some of data reported here were not normalized to measures of adiposity and included all 26 of the diabetic subjects compared with controls or all 26 of the diabetic subjects in each treatment group. In this respect the data in Tables 1Go and 2Go did not significantly differ if all 26 subjects, as opposed to 23 or 21, were included.

Plasma leptin is strongly related to percent body fat and is independent of gender

Among the combined nonobese and BMI-matched nondiabetic control groups, a strong curvilinear relationship was observed when leptin was plotted against percent body fat that fit well to an exponential curve. Subsequently, we noted strong correlations between log leptin and various measures of adiposity (Fig. 2Go). The strongest occurred when leptin was expressed relative to percent fat (Fig. 2AGo) as opposed to total body fat mass (Fig. 2BGo) or BMI (Fig. 2CGo). The relationship between log leptin and percent body fat appeared independent of gender. The linear curve of leptin vs. percent fat was used to calculate normalized leptin values expressed as percent predicted. This normalized value was calculated as 100 x the unmodified measured leptin concentration divided by the antilog of the predicted log leptin based on the linear relationship shown in Fig. 2AGo. Likewise, leptin values were normalized to total fat and BMI. As shown in Fig. 2DGo, leptin differed by gender when normalized to total fat or BMI. As Rosenbaum et al. (26) reported a strong linear correlation between unmodified plasma leptin concentration and fat mass, we also examined our data in this way and observed a similar strong relationship (Fig. 2EGo) and a difference in leptin/fat mass by gender (Fig. 2FGo).

***02**

In other analyses (not shown graphically) we examined log leptin as a function of percent truncal and percent nontruncal fat (percentage of total body mass composed of truncal or nontruncal fat). In this regard, DEXA does not separate intraabdominal truncal fat from sc truncal fat. Among the combined nonobese and BMI-matched nondiabetic groups, log leptin correlated to percent truncal fat (r2 = 0.664; P < 0.0001) and percent nontruncal fat (r2 = 0.597; P < 0.0001), but not as strongly as percent whole body fat (Fig. 2AGo). Leptin normalized to nontruncal fat (expressed as the percent predicted, calculated as described above) did not significantly differ by gender (102 ± 4 in females compared with 96 ± 7 in males), but differed when normalized to truncal fat (112 ± 4 in females compared with 80 ± 4 in males). Females had a higher percent truncal fat than males (17.0 ± 0.8 compared with 11.9 ± 1.0; P < 0.001, by unpaired t test).

Plasma leptin is decreased in diabetic subjects

Plasma leptin was plotted against percent body fat in the control (combined nonobese and BMI-matched groups) and diabetic subjects at baseline, and exponential curves were fit to both groups (Fig. 3AGo). When examined as an exponential function of percent body fat, plasma leptin is reduced at any given percent fat above 25%, the range in which our diabetic subjects were largely distributed (range, 22.1–56.8%, with only three subjects <26%). The difference between the control and diabetic subjects is evident in linear plots of log leptin vs. percent fat (Fig. 3BGo), wherein there is no overlap between the 95% confidence intervals for the slopes of these lines (slope for control subjects, 0.042; confidence interval, 0.036–0.047; r2 = 0.826; P < 0.0001 compared with a slope in the diabetic group of 0.027; confidence interval, 0.020–0.035; P < 0.0001). Subjects with IFG/mild DM were intermediate in normalized leptin per degree of percent fat (slope of leptin vs. percent fat, 0.035 ± 0.008; r2 = 0.688; P = 0.016).

****03*****

In another analysis we determined leptin normalized to percent fat, as described above using the control curve in Fig. 2AGo to determine the percent predicted leptin for both the control and diabetic subjects. The resulting values for normalized leptin were compared in the nonobese, BMI-matched, IFG/mild DM, and diabetic groups (Fig. 3CGo). This revealed a decrease in normalized leptin in the diabetic compared with the BMI-matched control group and an intermediate result for the IFG/mild DM group.

We further examined the effect of glucose intolerance and type 2 diabetes on plasma leptin in separate groups of male and female subjects. In these analyses, plasma leptin was expressed in alternative ways based on indexes of body fat. As indicated in Table 3Go, plasma leptin was significantly lower in female diabetic subjects compared with BMI-matched controls when normalized to any measure of adiposity. Table 3Go also shows that plasma leptin was lower in the male diabetic subjects compared with BMI-matched controls; however, those data did not achieve statistical significance. In this regard, we noted divergent relationships for log leptin or unmodified leptin concentration vs. indexes of adiposity (Fig. 3Go, B, D, and E). Thus, the greater adiposity among the female subjects may explain why the decrease in leptin secondary to diabetes (Table 3Go) achieved significance only for the female subjects.


Normalized leptin correlates to insulin secretory capacity

Insulin release and sensitivity were assessed using the insulin-modified FSIVGTT. Leptin normalized to percent fat correlated significantly with insulin release measured as the integrated area under the curve of glucose vs. time over 0–20 min of the FSIVGTT (insulin given at 20 min), but not to SI (Fig. 4Go). Leptin normalized to percent fat was poorly related to fasting plasma insulin (Fig. 4Go).
*********************

[Reza1969]

نوشته شده توسط silver

dear reza ... thanks for helping and improving our english knowledge..and congratulations for your EFl moderator post.. I have request for translating the below words ...I need them for a furniture web design

مبل استيل
2- مبل راحتي
3- بوفه ويترين
4- آينه کنسول
5- دراور
6- جلومبلي (عسلي)
7- پارتيشن
8- پيراهن مبل
9- رويه کوبي
10- ورق (mdf و فرميکا و ...)
11- ثرمه
12- معرق
13- برنز
14- منبت کاري
15- مصنوعات فلزي
16- جاکفشي

thanks a lot

Hi Silver

Sorry for the delay. I got the equivalents of some of the words(not all of them). I hope they help you out :

1. steel furniture
2. sofa
3. buffet
4.
5. drawer
6. coffee table
7. partition
8. furniture cover
9.
10. sheet(MDF,Formica,...)
11.
12.
13. bronze
14. fretwork
15. metal products
16. shoe cupboard

[Reza1969]

نوشته شده توسط Ship Storm

O Arab of the desert, I fear thou wilt not reach the Kaba Because the road on which thou travelest leads to Turkestan. Who is the poet of this couplet?

اي عرب بيابانگرد ، مي ترسم به كعبه نرسي زيرا اين راهي كه مي روي به تركستان است. اين بيت از كدام شاعر است؟

[*NashenaS*]

In another analysis we determined leptin normalized to percent fat, as described above using the control curve in Fig. 2AGo to determine the percent predicted leptin for both the control and diabetic subjects. The resulting values for normalized leptin were compared in the nonobese, BMI-matched, IFG/mild DM, and diabetic groups (Fig. 3CGo). This revealed a decrease in normalized leptin in the diabetic compared with the BMI-matched control group and an intermediate result for the IFG/mild DM group.

We further examined the effect of glucose intolerance and type 2 diabetes on plasma leptin in separate groups of male and female subjects. In these analyses, plasma leptin was expressed in alternative ways based on indexes of body fat. As indicated in Table 3Go, plasma leptin was significantly lower in female diabetic subjects compared with BMI-matched controls when normalized to any measure of adiposity. Table 3Go also shows that plasma leptin was lower in the male diabetic subjects compared with BMI-matched controls; however, those data did not achieve statistical significance. In this regard, we noted divergent relationships for log leptin or unmodified leptin concentration vs. indexes of adiposity (Fig. 3Go, B, D, and E). Thus, the greater adiposity among the female subjects may explain why the decrease in leptin secondary to diabetes (Table 3Go) achieved significance only for the female subjects.


Normalized leptin correlates to insulin secretory capacity

Insulin release and sensitivity were assessed using the insulin-modified FSIVGTT. Leptin normalized to percent fat correlated significantly with insulin release measured as the integrated area under the curve of glucose vs. time over 0–20 min of the FSIVGTT (insulin given at 20 min), but not to SI (Fig. 4Go). Leptin normalized to percent fat was poorly related to fasting plasma insulin (Fig. 4Go).

سلام دوست من....
اين انگليسي كه در اين متن به كار رفته يا كاملا تخصصي هست يا انگليسيش خيلي مسخرست!
من هيچي نمي فهمم... اما اين و فعلا بگير تا بعد...

در يه اناليز ديگه ما مشخص كرديم نسبت نرماليز لپاتين رو به چربي - همونطور كه در بالا به وسيله نمودار كنترل در گراف 2A بالا توضيح داده شده - براي مشخص كردن درصد پيش بيني شده براي كنترل و موضوعات ديابتيك!

[Dianella]

Hi dear Hamed
Here is the translation of second part of your text.
Hope it will be useful
have a nice day
bye

ترجمه بخش دوم:
در يك بررسي ديگر (كه به صورت نموداري نشان داده نشده است)‌ما ثبت لپتين را به عنوان عملكردي درصدي و
وابسته به تنه و همچنين درصد چربي غير تنه اي را آزموديم (درصد توده ي كل بدني از مجموع چربي بدني و
غير بدني به دست مي ايد). در اين مورد، دكسا سبب جدايي چربيهاي تنه اي بين شكمي از چربي تنه اي نمي
شود. در بين محتواي توده ي بدني گروه هاي غير ديابتي و nonobese هاي تركيبي لپتين ثبتي وابسته به درصد
چربي تنه اي (r2 = 0.664; P < 0.0001) و درصد چربي غير تنه اي (r2 = 0.597; P < 0.0001) مي باشد، اما اين
وابستگي به اندازه ي ميزان وابستگي آن به چربي كل بدني نيست. (شكل 2AGo). لپتين هنجار شده به چربي
غير تنه اي (بيان شده به صورت درصد مورد پيش بيني و محاسبه شده به روشي كه در زير آمده است) بر اساس
جنسيت فرد تغيير قابل ملاحظه اي نمي يابد (102+- 4 در زنان در مقايسه با 96+-7 در مردان)،‌در حاليكه همين
لپتين هنگامي كه با چربي تنه اي هنجار مي شود در دو جنس تفاوت قابل ملاحظه اي مي يابد (112+-4 در زنان و
80+-4 در مردان). همچنين بايد گفت كه زنان نسبت به مردان درصد چربي تنه اي بيشتري دارند (17.0 ± 0.8 در
مقايسه با 11.9 ± 1.0: P < 0.001، نتايج به دست آمده به وسيله ي آزمون تي غيرجفت)

در افراد مبتلا به ديابت ميزان لپتين پلاسما كاهش مي يابد
ميزان لپتين پلاسما در نمودار در برابر درصد چربي بدني در گروه شاهد (nonobese هاي تركيبي و گروه هاي داراي
محتواي توده بدني هماهنگ شده)‌ رسم شد كه در اين نمودار افراد مبتلا به ديابت در خط پايه قرار گرفتند و منحني
هاي نمايي با هر دو گروه تناسب يافتند. (شكل 3AGo).
لپتين موجود در پلاسما هنگامي كه به عنوان يك كاركرد نمايي از درصد چربي بدن مورد آزمون قرار گرفت،‌ در همه
ي درصدهاي چربي بالاي 25 درصد كاهش نشان داد، يعني در محدوده اي از ميزان چربي كه پراكندگي افراد
مبتلا به ديابت در آن بسيار زياد است. (ميزان چربي بدني افراد مبتلا به ديابت اكثرا در محدوده ي بالاي 25 درصد قرار
دارد). (محدوده ي 22.1–56.8% فقط بر اساس سنجش به عمل آمده از سه فرد <26%). تفاوت بين گروه شاهد و
افراد مبتلا به ديابت در نقشه هاي خطي لپتين ثبتي در برابر درصد چربي آشكار است (شكل 3BGo), در اين مورد
هيچ گونه همپوشاني بين فواصل قابل اطمينان 95 درصدي براي شيب اين خطوط وجود ندارد. (شيب منحني گروه
شاهد: 0.042،‌فاصله اطمينان 0.036–0.047، r2 = 0.826; P < 0.0001 در مقايسه به شيب منحني گروه
مبتلا به ديابت به مقدار 0.027، فاصله ي اطمينان 0.020–0.035; P < 0.0001). نمونه هاي همراه با IFG
/mild DM از لحاظ ميزان لپتين هنجار شده به ازاي هر درجه از درصد چربي در سطح متوسطي قرار داشتند. (
شيب منحني لپتين در برابر درصد چربي: 0.035 ± 0.008; r2 = 0.688; P = 0.016)

[*NashenaS*]

In another analysis we determined leptin normalized to percent fat, as described above using the control curve in Fig. 2AGo to determine the percent predicted leptin for both the control and diabetic subjects. The resulting values for normalized leptin were compared in the nonobese, BMI-matched, IFG/mild DM, and diabetic groups (Fig. 3CGo). This revealed a decrease in normalized leptin in the diabetic compared with the BMI-matched control group and an intermediate result for the IFG/mild DM group.

We further examined the effect of glucose intolerance and type 2 diabetes on plasma leptin in separate groups of male and female subjects. In these analyses, plasma leptin was expressed in alternative ways based on indexes of body fat. As indicated in Table 3Go, plasma leptin was significantly lower in female diabetic subjects compared with BMI-matched controls when normalized to any measure of adiposity. Table 3Go also shows that plasma leptin was lower in the male diabetic subjects compared with BMI-matched controls; however, those data did not achieve statistical significance. In this regard, we noted divergent relationships for log leptin or unmodified leptin concentration vs. indexes of adiposity (Fig. 3Go, B, D, and E). Thus, the greater adiposity among the female subjects may explain why the decrease in leptin secondary to diabetes (Table 3Go) achieved significance only for the female subjects.


Normalized leptin correlates to insulin secretory capacity

Insulin release and sensitivity were assessed using the insulin-modified FSIVGTT. Leptin normalized to percent fat correlated significantly with insulin release measured as the integrated area under the curve of glucose vs. time over 0–20 min of the FSIVGTT (insulin given at 20 min), but not to SI (Fig. 4Go). Leptin normalized to percent fat was poorly related to fasting plasma insulin (Fig. 4Go).

پست duplicate شد... ادامه ترجمه رو مجبورم بزارم..

نتايج لپتين نرماليزه شده در nonobese (نمي دونم چيه), BMI-matched, (منطبق با بي ام اي), IFG/mild DM و گروه هاي ديابتي (گراف 3CGo).
اين نشان داد (ما شاهد اين بوديم ) كه كاهش در مقدار عادي (نرماليزه) لپتين در يك شخص ديابتي در مقايسه با يك گروه كنترل BMI-matched... و يك نتيجه متعادل (متوسط) براي گروه IFG/mild DM .

ما تست هامون رو براي تاثير عدم تحمل گلوگز و ديابت نوع 2 روي لپتين پلاسما روي گروه هايي از مردان و زنان ادامه داديم... در اين اناليز ها لپتين پلاسما به شيوه هاي مختلف و بر اساس چربي موجود در بدن دسته بندي شد...
همون طور كه در جدول شماره ××× نشان داده شده مقدار معمول لپتين پلاسما در ديابت هاي زنانه كمتر از BMI-matched controls بوده .
جدول شماره 3Go همچنين نشون مي ده كه لپتين پلاسما در مردها هم كمتر از اونهايي هست كه به وسيله BMI-matched كنترل مي شن...

اگر چه اون داده ها (اطلاعات) از اهميت اماري خاصي بر خوردار نيستن!
در اين زمينه ما رابطه هايي صوقي (منظورم اينه كه به طرف يه چيزي نزول يا صعود مي كنه) رو براي ثبت لپتين يا غلظت هاي محلولي لپتين در مقايسه با ليست adiposity (نمي دونم ) (گراف هاي ×××) ثبت كرديم (برامون جالب بود).

در نتيجه مقدار بيشتر adiposity در ميان موردهاي زنانه مي تونه توضيح دهنده كاهش لپتين در ديابت نوع 2 (جدول ××) باشه و اين نتها در موردهاي زنانه به دست مياد.

لپتين معمولي به حجك قده ترشحي انسولين نسبت داده ميشه...
مقدار رها شدن انسولين و حساسيت اون توسط انسولين تقيير يافته FSIVGTT مورد ازمايش قرار گرفت. لپتين به درصدي از چربي كه به طور زيادي به رهاييه انسولين بستگي داشت به عنوان انتگرال مساحت زير منحني (گراف) گلوگز در مقابل زمان.. (يعني گلوكز توي y-axis قرار ميگيره و زمان در x-axis) به اندازه 0 تا 20 دقيقه ي FSIVGTT (انسوليني كه در 20 دقيقه داده شده) اما نه به واحد SI(گراف××)...
حالت نسبت متعادل لپتين به چربي خيلي كم مرتبط شده به (fasting رو نمي دونم به چه منظور اورده) انسولين پلاسما...

خوب بعد از 6 سال فارسي اينجوري حرف نزدن فكر كنم حسابي قلط داشته باشه... ديگه رضا جان گفتن منم نتونستم بگم نه...
اميدوارم مفيد باشه...
يا حق.